Can a keto diet help atherosclerosis

By | June 20, 2020

can a keto diet help atherosclerosis

A Michigan Medicine dietician explains how the popular low-carb regimen can get results — as well as related risks everyone should know. Touted by celebrities as a quick way to lose substantial weight, the ketogenic diet might seem counterintuitive to good heart health. Fruits, root vegetables, grain products and legumes all are prohibited. The intake is designed to trigger the metabolic state of ketosis, a process that occurs when the body burns off fat as an alternate source of energy. A keto diet can also lower elevated blood sugar linked to artery-damaging inflammation. Proteins comprise 20 percent — and carbohydrates make up just 5 percent. Cutting out low-quality carbs found in soft drinks and white bread, for instance, is a good idea for anyone, Ryskamp says. Sugar and starches raise the risk for obesity, diabetes and heart disease. Healthful, too, is a steady intake of green vegetables such as broccoli, celery, kale and spinach — all of which are permitted on the keto diet.

Updated Jun 9th, — Written by Craig Clarke. According to the World Health Organization, cardiovascular disease which is also commonly referred to as heart disease is the leading cause of death globally. On the surface, this sounds like a dreadful omen for humanity, but there is some sweetness to this bitter news once we realize that heart disease is preventable and, in some cases, reversible. Heart diseases are a group of disorders of the heart and blood vessels. In general, they fall into these categories. The most common, preventable, and reversible type of heart disease is coronary heart disease or coronary artery disease. This occurs when the arteries that supply blood to heart muscle become hardened and narrowed as plaque accumulates on their inner walls. The technical term for this is atherosclerosis.

The treatment of obesity and cardiovascular diseases is one of the most difficult and important challenges nowadays. Weight loss is frequently offered as a therapy and is aimed at improving some of the components of the metabolic syndrome. Results regarding the impact of such diets on cardiovascular risk factors are controversial, both in animals and humans, but some improvements notably in obesity and type 2 diabetes have been described. Unfortunately, these effects seem to be limited in time. Moreover, these diets are not totally safe and can be associated with some adverse events. Notably, in rodents, development of nonalcoholic fatty liver disease NAFLD and insulin resistance have been described.

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